How the immunologists made the ULTIMATE dumb mistake of thinking they would ONE UP the human body--NOT HAPPENING.

@johnconnor__err @Chris_CDC @tobyjrussell @dr_jon_l @vamosvigilante @thereal_truther @01Nov2022 @RandPaul

Essentially, for ANY foreign antigen, it is difficult to say HOW LONG it actually stays in the blood. As long as there are antibodies and its antigen in blood, why wouldn’t the PRECIPITINS be able to grow?

the J&J was a HUGE boost of antigen at ONE time. The mRNA, created spike spread out.

there is ALSO rate of clearance of immune complexes which will also vary tremendously from person to person. also depends on how FAST different people actually DO create antibodies. and remember, the FIRST antibody to come out? IgM. HUGE. image the precipitins with THOSE.

Isn't it just incredible? NONE of them even realized that the antibody will bind its antigen? for 200 years?

Now, with this COVID mRNA vaccine. Did the authors of the studies KNOW about PRECIPITINS? They HAD to have.

There are TWO options. They DID or didn't know.

IF these COVID mRNA researchers DID know about PRECIPITINS but decided NOT to check to see IF Precipitins would form in the blood of patients, they were involved in WILLFUL MISCONDUCT and TOTAL disregard for the health and safety of the public.

IF these COVID mRNA researchers DID NOT KNOW about PRECIPITINS, then they should IMMEDIATELY order the RECALL of EVERY COVID mRNA vaccine and ALERT the public to their MASSIVE OVERSIGHT.

And they should ACKNOWLEDGE to the world how DUMB they were NOT to know.

Of course, the other option is that they actually DID the research and DID test the blood for PRECIPITINS (antibody binding to its antigen, and why would it not).

So, if they DID DO the PRECIPITIN testing, let's see the research. Cough it up. ah, you do NOT have it?

Then, ONLY OPTION. Is to DO IT. AND BIG PHARMA.

PLEASE. Don't ask an EYE DOC to do it. This is YOUR mistake or willful misconduct. Correct it and BEFORE you have the peer reviewed published paper on the PRECIPITIN research, you do NOT know what the result will show.

(I would bet a MILLION dollars that the antibody and its antigen WILL clump together even in BLOOD).

Since you do NOT know what the PRECIPITIN research on your COVID vaccine will show, and since there is a HUGE chance that I AM correct, then don't you have to STOP the vax--

UNTIL you DO figure all this out????

Really. Your PITIFUL pro vax scientist DEFENDERS think it's MY JOB to do this PRECIPITIN research on YOUR VACCINE that YOU make the profits from. You agree? BIG PHARMA?? that THAT is NOT MY JOB??? LMAO.

WILL EAGERLY AWAIT THE RESULTS.

In science and medicine, if you don't ask the RIGHT questions, you will NEVER design the research trial CORRECTLY.

So, they can't JUST say, well, we DID check for "side effects."

One of my substack followers sent me info that shows that PRECIPITINS have been WELL STUDIED in SERUM.

Wow. That research I was supposed to do? Buy the COVID antibody? Buy the spike antigen? Use my blood and see if they cause PRECIPITINS in my blood?

Don't need to do it.

Serum is blood with cells and clotting factors removed.

IN SERUM, polyclonal antibodies and its antigen DO form PRECIPITINS. The noose is getting TIGHTER around the vaccine industry.

jiwaji.edu/pdf/ecourse/ph…(reference to a syllabus showing PRECIPITINS in serum.

It would be very difficult to detect PRECIPITINS if I put Polyclonal COVID antibodies and spike antigen into my blood because if whole blood is collected into a centrifuge tube without additives, then if left undisturbed, the blood will CLOT in 15-30 minutes.

The fact that visible PRECIPITINS form in SERUM that has NO CLOTTING factors is a much CLEANER experiment and no worries that the visible PRECIPITIN is actually a CLOT since there are NO CLOTTING FACTORS in SERUM.

DO I need to do ANYTHING MORE???

IF I had done the PRECIPITIN test in whole BLOOD, since BLOOD DOES clot when still at room temperature, it would have been HARD to interpret the results.

BUT, SERUM has NO clotting factors so the PRECIPITIN that is visible is CLEARLY NOT from clotting factors.

Now, the ONLY experiment left to do? THEY have NOT done either.

They have NOT shown IN VIVO, the COVID antibody binding a virus in the moving blood of a live patient.

I have NOT shown IN VIVO, the polyclonal COVID antibodies and its antigen forming PRECIPITINS in the moving blood of a live patient.

So, there IS no way to do that study.

and, in my life. I NEVER knew I could write SOOO much stuff on just this ONE topic.

When I started this four years ago? I would have NEVER believed that I would put out THIS MUCH STUFF.

And of course, I NEVER imagined I'd discover the STRING MECHANISM that would--

4 years ago? If you told me I would discover a mechanism that would destroy HALF the vaccines on earth? Yeah, I would have thought YOU were crazy.

But, here we are. To much fanfare. but just wait. The STRING will not die. It has a life of its own.

the most LOYAL follower I've ever had, is here to witness it.

And johnconnor who can't BELIEVE I write this fascinatingly interestingly when he reads my stuff is here too.

It's not like I'm a GENIUS because everything I predict about this STRING mechanism, other people keep finding evidence to perfectly support it.

it can seem like that and if I was a MORON Malone, I would just act like a genius and no one would question it later.

ONCE I thought, hey, this antibody might not even be NECESSARY in our fight against viruses? Once I made THAT decision?

All the facts I find, all the mechanisms I find, everything supports that FIRST decision.

meaning. If ANY NUMBER of intelligent people made --

--that SAME DECISION, to NOT believe in antibodies for viruses?

And if you KEEP looking for facts that SUPPORT your decision that antibodies aren't important in our fight against viruses?

You WILL KEEP FINDING THEM. ANYONE WOULD.

When I FIRST came to that conclusion in 2020, did I KNOW for absolute certain that I was correct? NO.

Could I PREDICT all the things I would find? NO.

So, I'm saying, when I find TWO separate mechanism that EACH destroy half the vaccines? NOT cuz I'm some CRAZY genius.

Because I MADE that decision to believe that ANTIBODIES were NOT useful in our fight against viruses.

When I FIRST made that decision? It was ONLY a BELIEF. So, IF my decision WAS CORRECT? I'm gonna KEEP FINDING STUFF and KEEP FINDING STUFF. Which I did.

My point is.

I'm NOT some crazy genius. I made the decision to believe that antibodies were NOT useful against viruses WITHOUT complete evidence.

Once I made THAT DECISION? And STUCK TO IT? I KEPT finding what ANYONE else would have KEPT finding.

My point is.

It is INFINITELY MORE LIKELY that the PARADIGM of the vaccine for viruses? Is WRONG than the likelihood that I'm this OUT OF THE WORLD GENIUS.

You see? McCullough? I wasn't even going to TAKE all that credit. I'm SMART ENOUGH. I'm SMART ENOUGH to know --

I'm SMART ENOUGH to know that I don't have those CRAZY formulas floating in my head, and I'm solving all these COMPLEX math equations and all that movie crap. NO. That doesn't happen in my head at all.

I'm SMART enough to know NOT TO LIE about how this happened in my head.

When I was previously on Twitter. they had a couple of celebration "victory lap" celebrations for me on some spaces. Both times I felt. why? I don't see it. I don't get it.

but, tonite? I'm finally sure.

Because of that finding, PRECIPITINS in SERUM.

and I have to include this very clever observation in this thread.

This one's a tiny bit deep. Now, the REASON the vaccinologists NEVER thought what would happen if they added antibody to antigen? would it not BIND? how come they didn't SEE this for 200 years?

because of the concept of "training."

Okay. so, the pathogen M is present. The antibody is made to M. The antibody sticks to pathogen M and destroys it. But, because the body has NO idea how MANY pathogen Ms are present, it makes MORE M antibodies. better to have MORE.

So, then the vaccinologists get confused and believe that MORE antibodies are created BECAUSE it was how the body TRAINED itself. No. NOT because the body was TRAINING ITSELF against the pathogen M.

The body made antibodies to pathogen M, not to TRAIN itself, but to DEFEAT it.

So, then why did the body make SO MANY M antibodies?

Because when the enemy could be ANYWHERE in the blood, and there's a LOT of blood, then you have to have MORE antibodies than PATHOGEN M if you're gonna GET all the PATHOGEN Ms.

and those b-lymphocytes? That are making M antibody? They HAVE to have a way of knowing if they need to make MORE M antibodies. So there's a feedback path available to LET the b-lymphocyte KNOW that they need to make MORE M antibodies for THIS CURRENT PATHOGEN M infection.

If there is a LOT of M antigen everywhere, then of course MORE B-lymphocytes will be making M antibodies & they ALL slow DOWN their production of M antibodies. BUT, if they DO get more M antigen on their surface, it's like, HEY GUYS, we have NOT won this WAR yet, we need MORE AB

Meaning. IT's not TRAINING, to be able to make MORE M antibodies. It's just the body's way of trying to win THIS WAR against PATHOGEN M. It's NOT for a LATER war. The body just wants to win THIS war against Pathogen M and how else would it do it? But have a feedback receptor?

and of COURSE that soldier, the M Ab making B lymphocyte doesn't just DIE OFF. That soldier STAYS ALIVE and can STILL work if that M antigen activates its M receptor. Again, he's an OLD soldier. contributes a LITTLE to the NEXT war. So what. That's not TRAINING.

Now, is it ALWAYS good? to destroy the M antigen when it is stuck on the M pathogen? ABSOLUTELY.

And that IS what happens.

To the point of this whole story. Vaccines.

Now becuz the immunologists THOUGHT that the M infection induced "training" and that the b lymphocytes were BETTER trained because of the M pathogen battle, they gave a person a vaccination of

M antigen. Then M antibodies were made. And IF the M antigen was STILL in the blood?

@johnconnor__err @Chris_CDC @tobyjrussell @dr_jon_l @vamosvigilante @thereal_truther @01Nov2022 @RandPaul @julie74439076 the body would go DESTROY whatever that M antibody was stuck to, in THIS case the M antibody sticks to the M antigen but no pathogen attached. JUST the M antigen.

But, the BODY STILL tries to destroy or CLEAR from the blood this M antibody stuck to this M antigen.

Remember, so far as the body is concerned? having a LOT of M antibodies when there isn't M antigen is pointless. When the body DOES make M antibody, if there still IS M antigen around? The M antibody will BIND to that M antigen and it's destroyed or cleared.

Now, the NEXT time that M pathogen somehow finds its way into the bloodstream, is the production of M antibody THAT much MORE and THAT much FASTER?

See, this is the PROBLEM with human minds. If there was a HUGE M-pathogen INFECTION, then LOTS of M antibodies WERE made.

Why is it TWO M infections? If it's ONE LONG war against the M pathogen, wouldn't you WANT the b lymphocytes to make more and more M antibodies?

It's NOT "training." "training" means that the FIRST time you did it? whatever you needed to be "trained" for?

You SUCKED at the activity. You CLEARLY showed you NEEDED "training."

But, we did NOT suck at fighting the M pathogen the first time we fought it. We won.

so, you ask, why do some B lymphocytes keep making M antibodies years later? Because it was a HUGE WAR against the M pathogen and they had HUGE numbers. BUT THE BODY WON. There ARE some B lymphocyte soldiers that live a LONG time and can STILL respond to M antigen and make Abs

Now, this HUGE WAR against the M pathogen. You SURVIVED IT. And it was a HUGE war. Did you B cells do JUST fine with NO TRAINING?

But why do you think you TRAINED your B cells? They did this AMAZING thing and made M antibodies having NEVER seen M pathogen.

Isn't that just INCREDIBLE? what the B lymphocytes did with NO TRAINING? They made this PERFECT M ANTIBODY that TAGS M ANTIGEN. Without ANY training, they DID THIS PERFECTLY. and becuz it was such a BIG war, there were LOTS of B lymphocytes that make M antibody.

sure, some of them live a LONG time and their positive feedback loops STILL work and they can STILL respond to M antigen to make some more M antibodies. But, they're OLD and TIRED. Let the YOUNG B lymphocytes make TONS of M antibodies.

and please remember. The YOUNG B lymphocytes have NEVER BEEN TRAINED to make M antibodies, YET, they do JUST fine. and since the NEXT huge M pathogen infection is a year away,

NEW young B lymphocytes which have NEVER made M antibody start PUMPING OUT M antibody. RIGHT?

For the SECOND M pathogen war, there will be NEW young B lymphocytes that have NEVER seen M antigen and YET do GREAT making M antibody, having NEVER been trained to make M antibody, RIGHT???

UMMM, where's your TRAINING AGAIN? No, you immunologists were CONFUSED.

Please. Don't pretend your vaccinations are "training."

Your vaccinations are NOT training. Your vaccinations are crying WOLF WOLF when there IS NO WOLF.

The M antigen is your vaccine. It goes into the body. The body makes M antibody. The M antibody complexes with M antigen

Your body is just going to try to get RID of all the M antigen it sees, and it will KEEP making M antibodies until it thinks this is DONE.

RIGHT??? So, SOMEHOW you all got it in your heads that because the second war, there was a bit more antibodies made, that it was TRAINING

No, I bet almost ALL the antibodies with the SECOND war against the M pathogen, i bet MOST of the antibodies were made by YOUNG b-lymphocytes that had NEVER even SEEN M antigen before and had NEVER been TRAINED, even if it IS the SECOND war.

again, don't just TALK about "training." BREAK IT DOWN. MOLECULE BY MOLECULE.

Explain to ME how the "training" occurs, just like I'm explaining to you stepwise, why i think there was NEVER any "training."

FOR one, the YOUNG B lymphocytes, make PERFECT antibodies the FIRST TIME

where's the training? Oh, you have a few OLDER M Ab making b lymphocytes STILL around from the FIRST war. So? Didn't the body HANDLE the first war? It HANDLED the first war with NO training, right???

To ALWAYS win the war against the M pathogen, the young B lymphocytes BETTER make the M antibody perfectly the FIRST time they ever make it, right?

And the B lymphocytes BETTER have a way of KNOWING if they need to make more M Abs, right?

and the B lymphocytes BETTER make a LOT more antibodies than there are M pathogens, RIGHT?

Because the M pathogens could be ANYWHERE. So you'll need a LOT more antibodies, RIGHT? AND, you BETTER get it RIGHT the first time or you're DEAD, RIGHT???

again, where's the training?

See, if the body didn't get it right the FIRST TIME? It would be DEAD and then you don't NEED to train the body, because it's dead, RIGHT?

But, if the body DID get it right, and the body DID win the war, it really doesn't need training, RIGHT?

So, if the BODY did NOT get it right the first time, and just DIED, you don't NEED to train a dead body.

And, if the BODY DID get it right the first time. the VERY first time, the body GOT IT RIGHT and made the PERFECT M antibody and PLENTY to spare, and FAST enough, really?

You can't just be IMPRESSED with the MIRACLE of what the BODY DID the FIRST time seeing the M pathogen? Making MORE than enough M antibodies using young B lymphocytes that had NEVER seen M antigen? Be impressed? Why are you so fascinated with TRAINING this amazing system????

The EGO of man is on display EVERYWHERE. For us to evolve THIS kind of intelligence? Then, our FUNDAMENTAL systems must be QUITE SOLID? Not needing TRAINING by our RECENTLY acquired awareness and intelligence? LMAO. You immunologists are QUITE the funny ones.

So, EVERY young B lymphocyte that sees its FIRST antigen, it almost ALWAYS makes the PERFECT antibody to the antigen. NEVER having been trained.

But, you silly immunologists want to TRAIN this INCREDIBLY efficient system???

Oh, and you OFFEND this incredible system.

You not ONLY INSIST to this amazing system

1) that you need to TRAIN IT,
2) you ACCUSE this system of having its timing COMPLETELY off.

Viral infections are raging at a week. Antibodies are BARELY ramping up at a week and max out at several weeks to a couple of months.

The PERFECT amazing system is using me to tell you guys to go UCK OFF.

It does NOT need "training" to take care of common bacterial infections.

it does NOT need "training" to take care of viral infections. this system is NOT off by WEEKS in producing antibody for viruses.

This b lymphocyte amazing antibody system? IS NOT USED by our body in its fight against viruses.

OTHERWISE, those who COULD make antibodies peak at a week would have a HUGE survival advantage over the rest of us 99.9% whose antibodies peak at around a month.

and then those early antibody peakers at a week would eventually be the majority. Oh, that isn't the case. ah, because antibodies aren't useful in our fight against viruses.

dummy immunologists. Better to assume the body is correct than to assume the body is stupid and tardy

well, the body asked me to tell you immunologists this.

”Okay. Yeah, Sorrrrryyy. We, the body, DID make antibodies against viruses. SO what. YOU immunologists, what would YOU do. Pathogen in blood. talk to the pathogen? Ask the pathogen if its a virus????

The body speaking through me. "there is NO WAY that I, a B LYMPHOCYTE, can EVER figure out if that pathogen is a virus or a bacteria. But, if I'm lazy and DO NOT make antibodies, if that pathogen happens to be a BACTERIA, and I DON'T make antibodies, this body could DIE.

the b lymphocyte still RAGING at the immunologists. “SO, whenever i see a pathogen? KNOWING I can't TELL if it's a virus (if i COULD tell, I would just CHILL if it as a virus) or a bacteria, I just have to GET TO WORK and PUMP out antibodies, okay?

Lymphocyte still, "SO I KNOW. My antibodies against viruses don't help at ALL. If I KNEW how to tell? I just wouldn't make antibodies against viruses. BUT, DUMB IMMUNOLOGIST, there is NO WAY FOR ME TO KNOW, so SORRY FOR FAKING YOU OUT and making you look SO DUMB

and that's why immunologists NEVER realized their most BASIC science, that an antibody WILL bind its antigen, EVEN in a person's blood, EVEN if they thought they designed the antibody to stick to ONLY the pathogen antigen. silly immunologists.

Silly immunologists. so EGO filled that they think they're going to one-up the body

No, the body did FINE the first WAR against M pathogen, DOES NOT NEED training.

No, the body was NOT 3 weeks late in delivering antibodies for viruses. antibodies aren't used in our virus fight

JUST from THIS alone, the average person should be able to tell that the BODY is INFINITELY more likely to be correct than SILLY immunologists,

all of whom want to RUN from me, except the REALLY stupid ones, they want to call ME names.

The STRING MECHANISM exposes the STUPIDITY of pro vaccine scientists. BUT, they're actually a LOT stupider than that even. yeah, sad but true.

It's NOT just that antibodies aren't useful at ALL in our fight against viruses. It's that antibodies are actually HARMFUL.

and SURE, we'll put up with the collateral damage/harm that antibodies cause IF the antibody is DESTROYING the pathogen along with causing a bit of collateral damage.

BUT, when there is NO PATHOGEN? and the antibody is causing COLLATERAL DAMAGE?? FOR WHAT? WHY? ego of man.

and this is how I rip off the sheep's clothing from these evil wolves. dumb pretty much ends up being evil.